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Dems' Health Care Reform Plans Would Include Abortion Coverage, Washington Times Opinion Piece States
As lawmakers work to pass health reform legislation, "few are talking about" the "essential question" of whether "health reform will force taxpayers to pay for abortions for the first time in 30 years," Family Research Council President Tony Perkins writes in a Washington Times opinion piece. According to Perkins, "the short answer is yes" because there is no "explicit provision" in any Democratic health plan that would "[p]revent taxpayer funding of abortions as part of the health care benefit Congress is considering"; avert "delays in health care services that result in the death of the patient waiting for care"; or allow health care providers "to refuse to participate in health care-related action that violates their conscience." Perkins continues that the House"s reform proposal would provide federal coverage for ""family planning," the well-worn buzz word that includes abortion unless specified to the contrary." He adds that "it would be naive to assume, unless there is an explicit prohibition in the bill, that [HHS] Secretary Kathleen Sebelius will not use her discretion to fund abortions with taxpayers" money." Perkins also writes that the Democratic reform plans, "in short, ... attempt to be silent on the key question of whether or not to allow the U.S. government to fund abortions with taxpayers" money," and also give the HHS secretary "the power to allow taxpayer-funded abortions."He writes, "The Family Research Council"s answer is clear: There must be a permanent prohibition on taxpayer-funded abortions," as well as "provision to allow a right of conscience for doctors and nurses and other health care providers" to refuse to participate in treatments they oppose. He adds that "there can be no system of denial or delay or rationing of care." Perkins concludes, "Euthanasia by any other name is a poison pill in the health reform debate" (Perkins, Washington Times, 7/5)
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Norovirus And Salmonella Were Leading Causes Of Foodborne Disease Outbreaks In 2006
Norovirus and Salmonella were the leading causes of foodborne disease outbreaks in 2006, according to a report released by the Centers for Disease Control and Prevention. The report, based on investigations of foodborne disease outbreaks provides the most recent report of how many illnesses were linked to specific types of foods.
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Meningitis Is A Serious Threat To Hajj / Umrah Travellers
Association of British Hujjaj (Pilgrims) UK a National Hajj Specific and charitable organisation warned British Hajj that they are at risk of infections including meningococcal disease because of crowded conditions at ceremonies, accommodation sites and on public transport.
Diagnostics

Prenatal Malaria Exposure Increases Risk Of Malaria And Anemia For Some Children

Some babies who are exposed to malaria before birth develop a tolerant phenotype that increases their susceptibility to malaria and anemia in childhood, says a new study in the open access journal PLoS Medicine. Indu Malhotra and Christopher King (Case Western Reserve University) and colleagues studied 586 newborns residing in a malaria-holoendemic area of Kenya to age three, assessing their malaria infection, malaria-specific immune responses, and anemia, and classifying them into three groups: "sensitized" babies in which cord blood cells made activating cytokines in response to malaria antigens; "exposed, not-sensitized" babies in which cord blood cells did not make activating cytokines but made an inhibitory cytokine (IL-10); and "not-exposed" babies born to uninfected mothers. The authors report that in the first 3 years of life, the exposed, not-sensitized newborns had a 60% greater risk of malaria infection than the unexposed group and a slightly higher risk of malaria infection than the sensitized group. They also had lower hemoglobulin levels, a sign of anemia, than the other babies. At 6 months, the T-cells of exposed, not-sensitized children were less likely to make activating cytokines in response to malaria antigens but made more IL-10 than the T-cells of the other children; malaria-specific antibody levels were similar in the three groups, say the authors. Why some children exposed to malaria before birth become tolerant to the disease while exposure to malaria antigens "primes" the immune system of other children to respond efficiently to malaria antigens is not clear. However, these findings could have important implications for the design of malaria vaccines for use in areas where children are often exposed to malaria before birth and for the design of strategies for the prevention of malaria during pregnancy, say the authors. "Once the significance of fetal malaria experience is better understood," the authors say, "it should translate into more effective strategies for malaria chemoprophylaxis during pregnancy." In a related Perspective on the study, Lars Hviid (not involved in the research) states that the research by Dr. King and colleagues "adds significantly to our understanding of prenatal exposure to P. falciparum antigens" and has "obvious clinical importance." But he outlines several areas for further investigation of pregnancy-associated malaria. Funding: The work was supported in part by United States Public Health Service grants I064687, AI054711, MH080601, AI065717 and Veterans" Affairs Research Service. The funders had no role in the study design, data collection, analysis, decision to publish or preparation of the manuscript. They did contribute to the design of the study. Competing Interests: David Narum has declared the following competing interest: ""Narum et al., United States Patent 7,078,507 for production of recombinant EBA-175 RII protein."" Citation: "Can Prenatal Malaria Exposure Produce an Immune Tolerant Phenotype?: A Prospective Birth Cohort Study in Kenya." Malhotra I, Dent A, Mungai P, Wamachi A, Ouma JH, et al. (2009) PLoS Med 6(7): e1000116. doi:10.1371/journal.pmed.1000116 PLoS Medicine


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