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Primary Insomnia Linked To A Neurochemical Abnormality
A research abstract presented on June 9, at SLEEP 2009, the 23rd Annual Meeting of the Associated Professional Sleep Societies, is the first demonstration of a specific neurochemical abnormality in adults with primary insomnia (PI), providing greater insight to the limited understanding of the condition"s pathology.

Influenza Monitoring By The US Military
The recent global swine flu outbreak has underscored the critical need for good surveillance and access to rapid epidemiological data. The US military, starting with early monitoring efforts in the 1970s, has developed a broad-based influenza monitoring system. In an article published in the September 2009 issue of the American Journal of Preventive Medicine, researchers describe this little-known national jewel that has repeatedly made notable contributions to global influenza control through close collaboration with CDC, the Food and Drug Administration (FDA), the WHO, and many other partners.
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Evaluating Language In Children With Autism
A new parent questionnaire, developed at the University of Waterloo, will help health practitioners to more accurately gauge the acquisition of language skills in children with autism.
Endocrinology

Comment On Dimebolin (dimebon) Increasing Brain Beta Amyloid In Mouse Models

Dimebolin, originally administered as an antihistamine, has previously been found to help slow the decline of cognitive ability in participants. This study analysed tau and how dimebolin affects build up of the toxic beta amyloid protein, which is widely believed to be linked to the development of dementia. This research showed that while dimebolin had a beneficial effect it actually caused an increase of the beta amyloid in mouse models. "This head scratching research has thrown up surprising results and suggests that while dimebolin provides cognitive benefits it may actually increase levels of toxic beta amyloid in the brain. One possible explanation may be that dimebolin is accelerating the process of beta amyloid development, which may reduce its toxicity. The questions surrounding whether beta amyloid causes nerve death in Alzheimer"s or is a bi-product of the process must be addressed by further research. "One in three people over 65 will die with dementia. Further, more robust research is needed to clarify whether dimebolin is an effective treatment for Alzheimer"s in the long term and if these puzzling increases in beta amyloid are also seen in human brains. With the right investment, dementia can be defeated. Alzheimer"s Society funds research to improve the lives of people today and to find a cure for tomorrow." Professor Clive Ballard Director of Research Alzheimer"s Society Reference: Samuel Gandy, Dimebon regulates amyloid beta metabolism in cultured cells, isolated nerve terminals and interstitial fluid of living rodent brain. Alzheimer"s Society


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